視黃酸, the molecular formula is C20H28O2, is a metabolic intermediate product of vitamin A in the body, which mainly affects the growth of bone and promotes epithelial cell proliferation, differentiation, keratolysis and other metabolic effects. For the treatment of acne vulgaris, psoriasis, ichthyosis, lichen planus, pityriasis rubra pilaris, keratosis pilaris, squamous cell carcinoma and melanoma.
視黃酸 had no effect on normal human melanocyte tyrosinase activity and melanin composition. When the skin undergoes physiological aging, or is damaged by drugs, ultraviolet radiation and trauma, Retinoic Acid can correct or prevent the abnormality of the biochemical composition and morphological structure of the dermal connective tissue caused by harmful factors, stimulate the synthesis of extracellular matrix protein in the skin, and be in the upper part of the dermis. Accelerates the formation of new bands of connective tissue and can increase the tensile strength of the wound site.
Retinoic Acid has no effect on normal skin collagen synthesis. In addition, Retinoic Acid has inhibitory activity on leukocyte chemotaxis, thereby exerting an anti-inflammatory effect. Retinoic Acid has no direct effect on sebaceous glands and their secretion.
視黃酸 had no effect on normal human melanocyte tyrosinase activity and melanin composition. When the skin undergoes physiological aging, or is damaged by drugs, ultraviolet radiation and trauma, Retinoic Acid can correct or prevent the abnormality of the biochemical composition and morphological structure of the dermal connective tissue caused by harmful factors, stimulate the synthesis of extracellular matrix protein in the skin, and be in the upper part of the dermis. Accelerates the formation of new bands of connective tissue and can increase the tensile strength of the wound site.
Retinoic Acid has no effect on normal skin collagen synthesis. In addition, Retinoic Acid has inhibitory activity on leukocyte chemotaxis, thereby exerting an anti-inflammatory effect. Retinoic Acid has no direct effect on sebaceous glands and their secretion.